Figure 4. (Table1 EEA) Multi-causality and Complexity - Figures and Tables

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Figure 4. (Table1 EEA) Multi-causality and Complexity
HOST GENETICS	+	HOST STATE	+	EXPOSURE/ DOSES	=	EFFECTS	=>	HARM
• Genotypes and Phenotypes		• Nutritional status • Immune status • Health status (lifest yle) • Age etc.		• Multiple exposures/doses • Via several exposure routes (skin, inhalation, ingestion) • Environmental- indoor • Environmental- outdoor • Occupational • Low doses and/or peaks • 24 hours continuous • Or single dose at critical times • Pre- and post-natal doses • Lifetime and cumulative •'Target' organ dose • Biologically effective dose • Often unknown doses		• Initiating • Promoting • Retarding • Suppressing • Causing Changes in • Cells, tissues, • Hormones • Organs • Functions; •'normal' distributions of bio- functions.		• Infections • Skin • Cancer • Neurotoxicity • Respirator y • Circulator y • Reproductive • Developmental health impacts • Other adverse

4-weekly data 1995-2000

Source: Data from CDSC www.phls.co.uk quoted in Baseline Report on Respiratory Health Section 2.1

Related publication:

Respiratory Diseases in Children

Other Figures & Tables on this publication:

Figure 1. Identifiable causes of death in children and young adults in Africa and South East Asia (ages 0-44)

Figure 2. Reports of Respiratory Syncitial Virus (RSV) isolates from 1995-2000

Figure 3a: Total workload in Scottish General (primary care) continuous morbidity reporting practices 2000 in the whole population showing the significant proportion of respiratory problems at all ages particularly in young children.

Figure 3b. Respiratory Disease workload per 1000 patients

Figure 4. (Table1 EEA) Multi-causality and Complexity

Figure 5: Multi-causality Framework for Environment and Health

Figure 6. Expression of asthma and hay fever in the main European regions